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Product Name: GAL3ST1 antibody [N1C3]
Applications: WB
Predicted Target Size: 49 kDa (note) (13)
Positive Controls: 293T , A431 , H1299 , HeLa , HepG2 , Molt-4 , Raji
Form Supplied: Liquid
Concentration: 0.91 mg/ml (Please refer to the vial label for the specific concentration)
Purification: Purified by antigen-affinity chromatography.
Full Name: galactose-3-O-sulfotransferase 1
Background: Sulfonation, an important step in the metabolism of many drugs, xenobiotics, hormones, and neurotransmitters, is catalyzed by sulfotransferases. The product of this gene is galactosylceramide sulfotransferase which catalyzes the conversion between 3-phosphoadenylylsulfate + a galactosylceramide to adenosine 3,5-bisphosphate + galactosylceramide sulfate. Activity of this sulfotransferase is enhanced in renal cell carcinoma. [provided by RefSeq]
Synonyms: CST antibody, GAL3ST1 antibody, cerebroside sulfotransferase antibody, galactosylceramide sulfotransferase antibody, 3-phosphoadenosine-5-phosphosulfate:GalCer sulfotransferase antibody, cerebroside (3-phosphoadenylylsulfate:galactosylceramide 3) sulfotransferase antibody, GalCer sulfotransferase antibody, 3-phosphoadenylylsulfate:galactosylceramide 3-sulfotransferase antibody, galactose-3-O-sulfotransferase 1 antibody
Cellular Localization: Golgi apparatus membrane; Single-pass type II membrane protein
CAS NO: 1402837-76-6
Product: MRT68921
Host: Rabbit
Clonality: Polyclonal
Isotype: IgG
Immunogen: Recombinant protein encompassing a sequence within the center region of human GAL3ST1. The exact sequence is proprietary.
Antigen Species: Human
Species Reactivity: Human
Conjugation: Unconjugated
Storage Buffer: 0.1M Tris, 0.1M Glycine, 10% Glycerol (pH7). 0.01% Thimerosal was added as a preservative.
Storage Instruction: Keep as concentrated solution. Aliquot and store at -20oC or below. Avoid multiple freeze-thaw cycles.
Notes: For In vitro laboratory use only. Not for any clinical, therapeutic, or diagnostic use in humans or animals. Not for animal or human consumption.
Specificity:
PubMed ID:http://www.ncbi.nlm.nih.gov/pubmed/25502723?dopt=Abstract

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But there are actually no intrachain backbone hydrogen bonds. Inside the solid state NMR derived
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Venging receptor on lymphatic endothelial cells. Especially, in the absence ofVenging receptor on lymphatic endothelial
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Formation. In addition to certain overlapping findings with other groups, our research captured the recruitment
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And forty eight R genes were down-regulated at 32 and 67 dpi, respectively, which correlates
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Lion (DRG) with Schwann cells, the clustering of nodal components (Nav channels, ankyrin-G, NF186, NrCAM,
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Roformate estersNIH-PA Author Manuscript NIH-PA Author ManuscriptCan Chem Trans. Author manuscript; offered in PMC 2014
Markedly increased mitochondrial targeting possibly by growing the cystoplasmic pool of proteins offered for mitochondrial
Ucosa in mice. Interestingly, in our mouse study, DAPM suppressed tumorUcosa in mice. Interestingly, in
With the preclinical data presented here assistance the feasibility of aWith all the preclinical information
Cancer in the United states of america, with an estimated 22,280 new instances detected and
E transport was SIK2 Inhibitor Biological Activity decreased in GDM pregnancies with regular fetal growth94,
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Tions related with antiviral resistance among diverse lineages.Author Manuscript Author Manuscript Author Manuscript Author Manuscript2.
Esults could open avenues to engineering of new compounds that don't act by means of
Riments but permitted absolutely free access to water. Rabbits were divided into 2 groups at
Observed for the H257R mutant [27]. The central role of protonationObserved for the H257R mutant
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